Czaroy
Active Member
Bonjour,
Je parcours régulièrement les forums US, et je souhaite vous partager la PCT made in Dave Palumbo, pour ceux qui ne connaissent pas le personnage, c'est un ancien bodybuildeur, un gourou des stéroïdes.
Q: What exactly does HCG, Clomid, and Nolvadex do in the body? I'm always reading on the message boards that it's imperative to use these three drugs, but I never really understood why. If I'm on a limited budget, can I use something like Arimidex instead of those three?
A: HCG (human chorionic gonadotropin) is a hormone that's isolated from the urine of pregnant women. HCG essentially mimics the pituitary hormone LH (luteinizing hormone); therefore, when it's injected into a male body, it stimulates the testicles to produce testosterone. Clomid (a synthetic estrogen) mimics the hypothalamic hormone GnRH (gonadrotropin-releasing hormone) and thus stimulates tha tpituitary gland to start producing LH and FSH. As previously mentioned, LH then stimulates the Leydig cells of the testicles to produce testosterone and FSH stimulates the Sertoli cells of the testicles to produce sperm. Aromatase inhimbitors such as Arimidex (anastrozole), Femara (letrozole), and Aromasin (exemestane) block the conversion of testosterone to estrogen. Estrogen management is extremely important during the post cycle period since it's estrogen (not testosterone) that shuts down hypothaamic-pituitary-testicular function. That's right, estrogen turns that entire process off! Therefore, once we turn on the hypothalamus (with Clomid) and the testcles (with HCG), we can keep them functioning by minimizing estrogen. Nolvadex (tamoxifen citrate) is and estrogen receptor antagonist. This means that Nolvadex blocks estrogen receptors on the surface of cells; it doens't stop the production of estrogen. As far as I'm concerned, this drug has no place in a steroid user's post-cycle therapy (PCT) since it does very little to help restore pituitary, hypothalamic, or testicular function. To make matters worse, Nolvadex has been shown to inhibit the release of the muscle-building peptide, IGF-1, from the liver. Following a heavy steroid cycle, HCG should be take post-cycle, at a dosage of 2,000 IU every third day for a total of five injections (two weeks). This protocol will awaken dormant testicular cells and it'll get them to start cranking out endogenous testosterone. Likewise, the HCG protocol should be followed up with a course of Clomid (100mg per day) for another two-week time period. Ideally, these two mini-cycles should be concluded with a four-six week cycle of a good anti-aromatase such as Arimidex (1mg every other day), Femara (2.5mg every other day), or Aromasin (25mg every other day).
Week 1 - 2 : 2,000 IU HCG E3D
Week 3 - 4 : 100mg/day Clomid
Week 5 - Week 10 : 0.5-1mg Adex EOD
J'aurais aimé avoir votre avis, car ça tiens LARGEMENT debout, et ça remettrais en cause pas mal de chose, comme l'utilisation du nolvadex en PCT. Et l'utilisation d'un anti-aromatase après la PCT, pour maintenir un axe HPTA stable, pendant une période non négligeable.
Je parcours régulièrement les forums US, et je souhaite vous partager la PCT made in Dave Palumbo, pour ceux qui ne connaissent pas le personnage, c'est un ancien bodybuildeur, un gourou des stéroïdes.
Q: What exactly does HCG, Clomid, and Nolvadex do in the body? I'm always reading on the message boards that it's imperative to use these three drugs, but I never really understood why. If I'm on a limited budget, can I use something like Arimidex instead of those three?
A: HCG (human chorionic gonadotropin) is a hormone that's isolated from the urine of pregnant women. HCG essentially mimics the pituitary hormone LH (luteinizing hormone); therefore, when it's injected into a male body, it stimulates the testicles to produce testosterone. Clomid (a synthetic estrogen) mimics the hypothalamic hormone GnRH (gonadrotropin-releasing hormone) and thus stimulates tha tpituitary gland to start producing LH and FSH. As previously mentioned, LH then stimulates the Leydig cells of the testicles to produce testosterone and FSH stimulates the Sertoli cells of the testicles to produce sperm. Aromatase inhimbitors such as Arimidex (anastrozole), Femara (letrozole), and Aromasin (exemestane) block the conversion of testosterone to estrogen. Estrogen management is extremely important during the post cycle period since it's estrogen (not testosterone) that shuts down hypothaamic-pituitary-testicular function. That's right, estrogen turns that entire process off! Therefore, once we turn on the hypothalamus (with Clomid) and the testcles (with HCG), we can keep them functioning by minimizing estrogen. Nolvadex (tamoxifen citrate) is and estrogen receptor antagonist. This means that Nolvadex blocks estrogen receptors on the surface of cells; it doens't stop the production of estrogen. As far as I'm concerned, this drug has no place in a steroid user's post-cycle therapy (PCT) since it does very little to help restore pituitary, hypothalamic, or testicular function. To make matters worse, Nolvadex has been shown to inhibit the release of the muscle-building peptide, IGF-1, from the liver. Following a heavy steroid cycle, HCG should be take post-cycle, at a dosage of 2,000 IU every third day for a total of five injections (two weeks). This protocol will awaken dormant testicular cells and it'll get them to start cranking out endogenous testosterone. Likewise, the HCG protocol should be followed up with a course of Clomid (100mg per day) for another two-week time period. Ideally, these two mini-cycles should be concluded with a four-six week cycle of a good anti-aromatase such as Arimidex (1mg every other day), Femara (2.5mg every other day), or Aromasin (25mg every other day).
Week 1 - 2 : 2,000 IU HCG E3D
Week 3 - 4 : 100mg/day Clomid
Week 5 - Week 10 : 0.5-1mg Adex EOD
J'aurais aimé avoir votre avis, car ça tiens LARGEMENT debout, et ça remettrais en cause pas mal de chose, comme l'utilisation du nolvadex en PCT. Et l'utilisation d'un anti-aromatase après la PCT, pour maintenir un axe HPTA stable, pendant une période non négligeable.